Pam Kreeger

From OpenWetWare

(Difference between revisions)
Jump to: navigation, search
Line 18: Line 18:
The objective of my research is to develop quantitative models of how normal and neoplastic OSE behave.  In particular, I seek to identify how EGF signaling impacts proliferation and invasive behavior of normal OSE and ovarian cancer cells.
The objective of my research is to develop quantitative models of how normal and neoplastic OSE behave.  In particular, I seek to identify how EGF signaling impacts proliferation and invasive behavior of normal OSE and ovarian cancer cells.
 +
• [[Lauffenburger:PamKreeger|Previous Research]]
• [[Lauffenburger:PamKreeger|Previous Research]]

Revision as of 09:30, 12 April 2006

Lauffenburger Lab

Image:wikiburger_top.jpg

Home        People        Research        Publications        Links        Internal       

Pamela K. Kreeger (BE postdoctoral)


Ph.D. Northwestern University, Chemical Engineering, 2005

B.S. Valparaiso University, Chemistry, 2000


Ovarian cancer is the deadliest of all gynecological cancers, with a mortality rate of nearly 60%. Currently, ovarian cancer is associated with a poor prognosis as the majority of patients are diagnosed only after the disease has spread into the abdominal cavity. Nearly 90% of ovarian tumors originate in the ovarian surface epithelium (OSE), a thin layer of cells covering the ovary that has no known major function. Importantly, there is limited understanding of the precursor lesion and tumor progression, inhibiting the development of diagnostic tests and effective treatments.

The objective of my research is to develop quantitative models of how normal and neoplastic OSE behave. In particular, I seek to identify how EGF signaling impacts proliferation and invasive behavior of normal OSE and ovarian cancer cells.


Previous Research

Curriculum Vitae

Personal tools