Qianben Wang Laboratory: Difference between revisions
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<center><font face="lucida fax" style= "color: | <center><font face="lucida fax" style= "color:Yellow" size="+10">'''Qianben Wang Laboratory'''</font> | ||
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<center><font face="lucida fax" style= "color: | <center><font face="lucida fax" style= "color:Yellow" size="+2">'''Laboratory of Cancer Epigenomics'''</font></center> | ||
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<center><font face="lucida fax" style= "color:Yellow" size="+1">'''Department of Molecular and Cellular Biochemistry, The Ohio State University'''</font> | |||
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[[Wang | <font face="lucida fax" style="color:#ffffff" size="+1"> '''Home''' </font>]] | [[Wang | <font face="lucida fax" style="color:#ffffff" size="+1"> '''Home''' </font>]] | ||
[[Wang:Research | <font face="lucida fax" style="color:#ffffff" size="+1"> '''Research''' </font>]] | [[Wang:Research | <font face="lucida fax" style="color:#ffffff" size="+1"> '''Research''' </font>]] | ||
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<font face="lucia fax">''' | [[Image:DOD 3.jpg]] | ||
<div style="text-align:left" font face="lucia fax">'''A histone modification governs androgen receptor to bind to regulatory regions of a class of cell cycle genes in castration-resistant prostate cancer but not in androgen-dependent prostate cancer. This leads to over-expression of cell cycle genes and accelerated growth of castration resistant prostate cancer. | |||
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Revision as of 12:57, 10 November 2011
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A histone modification governs androgen receptor to bind to regulatory regions of a class of cell cycle genes in castration-resistant prostate cancer but not in androgen-dependent prostate cancer. This leads to over-expression of cell cycle genes and accelerated growth of castration resistant prostate cancer.
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