Qianben Wang Laboratory

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<center><font face="lucida fax" style= "color:Crimson" size="+10">'''Qianben Wang Laboratory'''</font>
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<center><font face="lucida fax" style= "color:Yellow" size="+10">'''Qianben Wang Laboratory'''</font>
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<center><font face="lucida fax" style= "color:Crimson" size="+2">'''Laboratory of Cancer Epigenomics'''</font></center>
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<center><font face="lucida fax" style= "color:Yellow" size="+2">'''Laboratory of Cancer Epigenomics'''</font></center>
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<center><font face="lucida fax" style= "color:Yellow" size="+1">'''Department of Molecular and Cellular Biochemistry, The Ohio State University'''</font>
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[[Wang | <font face="lucida fax" style="color:#ffffff" size="+1"> '''Home''' </font>]] &nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;
[[Wang | <font face="lucida fax" style="color:#ffffff" size="+1"> '''Home''' </font>]] &nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;
[[Wang:Research | <font face="lucida fax" style="color:#ffffff" size="+1"> '''Research''' </font>]] &nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;
[[Wang:Research | <font face="lucida fax" style="color:#ffffff" size="+1"> '''Research''' </font>]] &nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;&nbsp;
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<div style="text-align:left" font face="lucia fax">'''Our laboratory focuses on studying the role of Androgen Receptor in prostate cancer.'''
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<div style="text-align:left" font face="lucia fax">'''A histone modification governs androgen receptor to bind to regulatory regions of a class of cell cycle genes in castration-resistant prostate cancer but not in androgen-dependent prostate cancer.  This leads to over-expression of cell cycle genes and accelerated growth of castration resistant prostate cancer.  
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Current revision

Qianben Wang Laboratory


Laboratory of Cancer Epigenomics


Department of Molecular and Cellular Biochemistry, The Ohio State University


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Image:DOD 3.jpg

A histone modification governs androgen receptor to bind to regulatory regions of a class of cell cycle genes in castration-resistant prostate cancer but not in androgen-dependent prostate cancer. This leads to over-expression of cell cycle genes and accelerated growth of castration resistant prostate cancer.
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