User:Andrew S. Gilder: Difference between revisions

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==Contact Info==
==Contact Info==
[[Image:OWWEmblem.png|thumb|right|Andrew S. Gilder (an artistic interpretation)]]
[[Image:OWWEmblem.png|thumb|right|Andrew S. Gilder (an artistic interpretation)]]


*Andrew S. Gilder
*Andrew Gilder
*University of Mississippi Medical Center
*UC San Diego
*Address 1
*Department of Pathology
*Address 2
*9500 Gilman Dr.
*City, State, Country etc.  
*La Jolla, CA 92093
*agilder@ucsd.edu
*[[Special:Emailuser/Andrew S. Gilder|Email me through OpenWetWare]]
*[[Special:Emailuser/Andrew S. Gilder|Email me through OpenWetWare]]


I work in the [[Your Lab]] at XYZ University. I learned about [[OpenWetWare]] from web, and I've joined because lab website.
https://www.researchgate.net/profile/Andrew_Gilder/
 
http://scholar.google.com/citations?user=MsyZ9MgAAAAJ
 
 
 
Currently a postdoctoral fellow at UC San Diego.
 
[[Hebert Lab]] Alumnus (2011) [http://biochemistry.umc.edu/hebert.html].


==Education==
==Education==
<!--Include info about your educational background-->
<!--Include info about your educational background-->
* Year, PhD, Institute
 
* Year, MS, Institute
* 2007, B.S. Biology, Economics Millsaps College
* Year, BS, Institute
* 2010, M.S. Biomedical Sciences, University of Mississippi Medical Center
* 2011, Ph.D. Biochemistry, University of Mississippi Medical Center


==Research interests==
==Research interests==
<!-- Feel free to add brief descriptions to your research interests as well -->
<!-- Feel free to add brief descriptions to your research interests as well -->
# Interest 1
# Control of gene transcription and splicing
# Interest 2
# Nuclear body dynamics
# Interest 3
# Epigenetics
# DNA damage response
# Cancer cell biology


==Publications==
==Publications==
<!-- Replace the PubMed ID's ("pmid=#######") below with the PubMed ID's for your publications.  You can add or remove lines as needed -->
<biblio>
#Paper1 pmid=6947258
#Paper2 pmid=13718526
// leave a comment about a paper here
#Book1 isbn=0879697164
</biblio>


==Useful links==
Mantuano E, Brifault C, Lam MS, Azmoon P, Gilder AS, Gonias SL. LDL
*[[OpenWetWare:Welcome|Introductory tutorial]]
receptor-related protein-1 regulates NFκB and microRNA-155 in macrophages to
*[[Help|OpenWetWare help pages]]
control the inflammatory response.
Proc Natl Acad Sci U S A. 2016 Jan 19. pii:
201515480. PubMed PMID: 26787872.
[http://www.pnas.org/content/early/2016/01/14/1515480113.long]
 
Gilder AS, Jones KA, Hu J, Wang L, Chen CC, Carter BS, Gonias SL. Soluble uPAR
is released selectively by glioblastoma cells that express EGF receptor variant
III and promotes tumor cell migration and invasion.
J Biol Chem. 2015 Apr 2. pii:jbc.M115.637488. [Epub ahead of print] PubMed PMID: 25837250.  [http://www.jbc.org/content/early/2015/04/02/jbc.M115.637488.long]
 
Hu J, Jo M, Eastman BM, Gilder AS, Bui JD, Gonias SL. uPAR induces expression
of transforming growth factor β and interleukin-4 in cancer cells to promote
tumor-permissive conditioning of macrophages.
Am J Pathol. 2014 Dec;184(12):3384-93.
doi: 10.1016/j.ajpath.2014.08.003. Epub 2014 Oct 11. PubMed
PMID: 25310970; PubMed Central PMCID: PMC4258500. [http://www.sciencedirect.com/science/article/pii/S000294401400488X]
 
 
Coilin participates in the suppression of RNA polymerase I in response to cisplatin induced DNA damage. Mol Biol Cell. 2011 April 1; Vol. 22(7):1070-1079,  PMID: 21289084 [http://www.molbiolcell.org/cgi/content/abstract/22/7/1070]Cover art: [http://www.molbiolcell.org/content/vol22/issue7/cover.shtml]
 
 
 
Cajal body formation correlates with differential coilin phosphorylation in primary and transformed cell lines. J Cell Sci. 2009 Jun 1;122(Pt 11):1872-81. PMID: 19435804  [http://jcs.biologists.org/cgi/content/abstract/122/11/1872?maxtoshow=&hits=10&RESULTFORMAT=&author1=hearst&andorexactfulltext=and&searchid=1&FIRSTINDEX=0&sortspec=relevance&resourcetype=HWCIT]
 
 
Relationship of the Cajal Body to the Nucleolus. ''The Nucleolus''. M.O.J. Olson (ed.).
Protein Reviews, 2011, Volume 15, Part 3, 361-380, DOI: 10.1007/978-1-4614-0514-6_16, Springer 2011.
[http://www.springerlink.com/content/t5u035538g136547/]
 
 
Fem1b promotes ubiquitylation and suppresses transcriptional activity of Gli1, Biochemical and Biophysical Research Communications, Available online 25 September 2013, ISSN 0006-291X, [http://www.sciencedirect.com/science/article/pii/S0006291X13015830]

Latest revision as of 15:05, 25 January 2016

Contact Info

Andrew S. Gilder (an artistic interpretation)

https://www.researchgate.net/profile/Andrew_Gilder/

http://scholar.google.com/citations?user=MsyZ9MgAAAAJ


Currently a postdoctoral fellow at UC San Diego.

Hebert Lab Alumnus (2011) [1].

Education

  • 2007, B.S. Biology, Economics Millsaps College
  • 2010, M.S. Biomedical Sciences, University of Mississippi Medical Center
  • 2011, Ph.D. Biochemistry, University of Mississippi Medical Center

Research interests

  1. Control of gene transcription and splicing
  2. Nuclear body dynamics
  3. Epigenetics
  4. DNA damage response
  5. Cancer cell biology

Publications

Mantuano E, Brifault C, Lam MS, Azmoon P, Gilder AS, Gonias SL. LDL receptor-related protein-1 regulates NFκB and microRNA-155 in macrophages to control the inflammatory response. Proc Natl Acad Sci U S A. 2016 Jan 19. pii: 201515480. PubMed PMID: 26787872. [2]

Gilder AS, Jones KA, Hu J, Wang L, Chen CC, Carter BS, Gonias SL. Soluble uPAR is released selectively by glioblastoma cells that express EGF receptor variant III and promotes tumor cell migration and invasion. J Biol Chem. 2015 Apr 2. pii:jbc.M115.637488. [Epub ahead of print] PubMed PMID: 25837250. [3]

Hu J, Jo M, Eastman BM, Gilder AS, Bui JD, Gonias SL. uPAR induces expression of transforming growth factor β and interleukin-4 in cancer cells to promote tumor-permissive conditioning of macrophages. Am J Pathol. 2014 Dec;184(12):3384-93. doi: 10.1016/j.ajpath.2014.08.003. Epub 2014 Oct 11. PubMed PMID: 25310970; PubMed Central PMCID: PMC4258500. [4]


Coilin participates in the suppression of RNA polymerase I in response to cisplatin induced DNA damage. Mol Biol Cell. 2011 April 1; Vol. 22(7):1070-1079, PMID: 21289084 [5]Cover art: [6]


Cajal body formation correlates with differential coilin phosphorylation in primary and transformed cell lines. J Cell Sci. 2009 Jun 1;122(Pt 11):1872-81. PMID: 19435804 [7]


Relationship of the Cajal Body to the Nucleolus. The Nucleolus. M.O.J. Olson (ed.). Protein Reviews, 2011, Volume 15, Part 3, 361-380, DOI: 10.1007/978-1-4614-0514-6_16, Springer 2011. [8]


Fem1b promotes ubiquitylation and suppresses transcriptional activity of Gli1, Biochemical and Biophysical Research Communications, Available online 25 September 2013, ISSN 0006-291X, [9]

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