User:Tkadm30/Notebook/Brainstorming: Difference between revisions
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* <font color=red>Cannabinoid agonists and the cholinergic system: Selective affinity for the mAChR receptor may indicates endogenous endocannabinoids as potential agents for neuroprotection agaisn't [http://www.sciencedaily.com/releases/2012/12/121202113712.htm organophosphate-derived] nerve gas precursors and Alzheimer disease.</font> (EC 3.1.1.8) | * <font color=red>Cannabinoid agonists and the cholinergic system: Selective affinity for the mAChR receptor may indicates endogenous endocannabinoids as potential agents for neuroprotection agaisn't [http://www.sciencedaily.com/releases/2012/12/121202113712.htm organophosphate-derived] nerve gas precursors and Alzheimer disease.</font> (EC 3.1.1.8) | ||
* Key roles for nanoparticles based chemical inducers and repressors are multiples and under intense research including but not limited to chemotherapy and bioinformatics. | * Key roles for nanoparticles based chemical inducers and repressors are multiples and under intense research including but not limited to chemotherapy and bioinformatics. | ||
* Endocannabinoid transport | |||
** retrograde 2-AG signaling may promote glutamatergic LTP. [https://www.ncbi.nlm.nih.gov/pubmed/23307660 PMID] | |||
Revision as of 06:14, 5 July 2015
- Important secondary messengers: NMDA, Dopamine, Serotonin, GABA, AChr, calmodulin, p38 MAP kinase
- Sites of interests: p38 (MEK), EGFR (Fyn), c-Src homolog domain (brain/hippocampus/amygdala), Ras/Raf/MEK pathway (Bcl-2), NMDAR, NRG1
- Cannabinoid agonists and the cholinergic system: Selective affinity for the mAChR receptor may indicates endogenous endocannabinoids as potential agents for neuroprotection agaisn't organophosphate-derived nerve gas precursors and Alzheimer disease. (EC 3.1.1.8)
- Key roles for nanoparticles based chemical inducers and repressors are multiples and under intense research including but not limited to chemotherapy and bioinformatics.
- Endocannabinoid transport
- retrograde 2-AG signaling may promote glutamatergic LTP. PMID
