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=== Synaptic hypercomputation ===
=== Synaptic hypercomputation ===


The Synaptic Hypercomputation (SH) hypothesis states that the phase coherence of neural communication may emerges via synaptic connectivity. This non-classical neurocomputational model is controlled by [[User:Etienne_Robillard/Notebook/Synaptic_exocytosis|synaptic exocytosis]], regulating neural synchronicity in the CNS.
The Synaptic Hypercomputation (SH) hypothesis states that the phase coherence of neural communication (synaptic plasticity) may emerges via long-range synchrony in the gamma range. This non-classical neurocomputational model is controlled by [[User:Etienne_Robillard/Notebook/Synaptic_exocytosis|synaptic exocytosis]], regulating neural communication in the brain.
 
Review: [http://www.sciencedirect.com/science/article/pii/S0896627315008235 Rhythms for Cognition: Communication through Coherence]


=== Pharmacological hypercomputation ===
=== Pharmacological hypercomputation ===


* Is pharmacological hypercomputation (PH) a dopamine-mediated synaptic function? (exocytosis)  
* Is pharmacological hypercomputation (PH) a dopamine-mediated synaptic function? (exocytosis)  
* Cannabinoids may rewire the brain connectivity by enhancing functional NMDA expression and [http://www.thenakedscientists.com/forum/index.php?topic=68779 cognitive intelligence].
* THC may rewire the brain connectivity by enhancing functional NMDA expression. (Reference needed)


== Neuronal phase coherence and transitions ==
== Neuronal phase coherence and synaptic transitions ==


=== Heteromeric transactivation of adenosine-CB1 receptors ===
=== Heteromeric transactivation of adenosine-CB1 receptors ===

Revision as of 17:41, 22 January 2017

Hypercomputation

Synaptic hypercomputation

The Synaptic Hypercomputation (SH) hypothesis states that the phase coherence of neural communication (synaptic plasticity) may emerges via long-range synchrony in the gamma range. This non-classical neurocomputational model is controlled by synaptic exocytosis, regulating neural communication in the brain.

Review: Rhythms for Cognition: Communication through Coherence

Pharmacological hypercomputation

  • Is pharmacological hypercomputation (PH) a dopamine-mediated synaptic function? (exocytosis)
  • THC may rewire the brain connectivity by enhancing functional NMDA expression. (Reference needed)

Neuronal phase coherence and synaptic transitions

Heteromeric transactivation of adenosine-CB1 receptors

How adenosine-CB1 heteromeric transactivation potentiate synaptic hypercomputation?

Neurexin-mediated trans-synaptic exocytosis

Could retrograde signaling promote synaptic proteins synthesis ? https://www.ncbi.nlm.nih.gov/pubmed/17035546

Discussion

See also