User:Tkadm30/Notebook/Hypercomputation: Difference between revisions

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== Neuronal phase coherence and synaptic transitions ==
== Neuronal phase coherence and synaptic transitions ==


=== Heteromeric transactivation of adenosine-CB1 receptors ===
=== Heteromeric transactivation of dopamine-CB1 receptors ===
How adenosine-CB1 heteromeric transactivation potentiate synaptic hypercomputation?
* How dopamine-CB1 heteromeric transactivation potentiate synaptic hypercomputation in the gamma band?
* Review: [http://molpharm.aspetjournals.org/content/67/5/1697.short Concurrent Stimulation of Cannabinoid CB1 and Dopamine D2 Receptors Enhances Heterodimer Formation: A Mechanism for Receptor Cross-Talk?]


===Neurexin-mediated trans-synaptic exocytosis===
===Neurexin-mediated trans-synaptic exocytosis===

Revision as of 03:40, 24 January 2017

Hypercomputation

Synaptic hypercomputation

The Synaptic Hypercomputation (SH) hypothesis states that the phase coherence of neural communication (synaptic plasticity) may emerges via long-range synchrony in the gamma range. This non-classical neurocomputational model is controlled by synaptic exocytosis, regulating neural communication in the brain.

Review: Rhythms for Cognition: Communication through Coherence

Pharmacological hypercomputation

  • Is pharmacological hypercomputation (PH) a dopamine-mediated synaptic function? (exocytosis)
  • THC may rewire the brain connectivity by enhancing functional NMDA expression. (Reference needed)

Neuronal phase coherence and synaptic transitions

Heteromeric transactivation of dopamine-CB1 receptors

Neurexin-mediated trans-synaptic exocytosis

Could retrograde signaling promote synaptic proteins synthesis ? https://www.ncbi.nlm.nih.gov/pubmed/17035546

Discussion

See also