User:Tkadm30/Notebook/Hypercomputation: Difference between revisions
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=== Heteromeric transactivation of adenosine-CB1 receptors === | === Heteromeric transactivation of adenosine-CB1 receptors === | ||
How adenosine-CB1 heteromeric transactivation potentiate synaptic hypercomputation? | How adenosine-CB1 heteromeric transactivation potentiate synaptic hypercomputation? | ||
===Neurexin-mediated trans-synaptic exocytosis=== | |||
Could retrograde signaling promote synaptic proteins synthesis ? https://www.ncbi.nlm.nih.gov/pubmed/17035546 |
Revision as of 03:38, 4 November 2016
Hypercomputation
Is neuronal hypercomputation a form of synaptic quantum tunnelling?
What is Biological Hypercomputation (BH)?
What is the speed of consciousness?
Is self-organized criticality (SOC) an evidence of biological hypercomputation?
Synaptic hypercomputation
The Synaptic Hypercomputation (SH) hypothesis states that the continuous phase transition of conscious states evolve via discrete computations inside neurons. This non-classical neurocomputational model is controlled by synaptic exocytosis, generating neural criticality in the CNS.
What is synaptic hypercomputation?
What is biological phase coherence?
Pharmacological hypercomputation
Is pharmacological hypercomputation (PH) a neurogenesis-mediated synaptic function? Cannabinoids may rewire the brain connectivity by enhancing functional neurogenesis and cognitive intelligence.
Heteromeric transactivation of adenosine-CB1 receptors
How adenosine-CB1 heteromeric transactivation potentiate synaptic hypercomputation?
Neurexin-mediated trans-synaptic exocytosis
Could retrograde signaling promote synaptic proteins synthesis ? https://www.ncbi.nlm.nih.gov/pubmed/17035546