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=== Neuronal phase coherence and synchronicity === | === Neuronal phase coherence and synchronicity === | ||
Neuronal phase coherence is "quantum-like" because long-range synchronicity is critical for optimal communication | Neuronal phase coherence is "quantum-like" entanglement because long-range synchronicity is critical for optimal communication | ||
in the gamma band. <cite>Paper3</cite> | in the gamma band. <cite>Paper3</cite> | ||
Revision as of 04:49, 5 March 2017
Hypercomputation
Synaptic hypercomputation
The Synaptic Hypercomputation (SH) hypothesis states that the phase coherence of neural communication (synaptic plasticity) may emerges via long-range synchrony in the gamma range. This non-classical neurocomputational model is controlled by synaptic exocytosis, regulating neural communication in the brain. [1]
Pharmacological hypercomputation
- Pharmacological hypercomputation (PH): a dopamine-CB1 cross-talk?
- Review: GPCR receptor heteromerization
- Heteromeric transactivation of dopamine-CB1 receptors:
- Dopamine-CB1 heteromeric transactivation may potentiate synaptic hypercomputation in the gamma band. [2]
Neuronal phase coherence and synchronicity
Neuronal phase coherence is "quantum-like" entanglement because long-range synchronicity is critical for optimal communication in the gamma band. [3]
Discussion
- Is neuronal hypercomputation a form of synaptic quantum tunnelling?
- What is biological hypercomputation?
- Is self-organized criticality (SOC) an evidence of biological hypercomputation?
- What is synaptic hypercomputation?
- Is synaptic hypercomputation a function of exocytosis?
- What is biological phase coherence?
- Neuronal phase coherence and synchronicity
References
-
Rhythms for Cognition: Communication through Coherence
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Concurrent Stimulation of Cannabinoid CB1 and Dopamine D2 Receptors Enhances Heterodimer Formation: A Mechanism for Receptor Cross-Talk?
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Phase-Coherence Transitions and Communication in the Gamma Range between Delay-Coupled Neuronal Populations