User:Tkadm30/Notebook/Hypercomputation: Difference between revisions
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The Synaptic Hypercomputation (SH) hypothesis states that the phase coherence of neural communication (synaptic plasticity) may emerges via long-range synchrony in the gamma range. This non-classical neurocomputational model is controlled by [[User:Etienne_Robillard/Notebook/Synaptic_exocytosis|synaptic exocytosis]], regulating neural communication in the brain. <cite>Paper1</cite> | The Synaptic Hypercomputation (SH) hypothesis states that the phase coherence of neural communication (synaptic plasticity) may emerges via long-range synchrony in the gamma range. This non-classical neurocomputational model is controlled by [[User:Etienne_Robillard/Notebook/Synaptic_exocytosis|synaptic exocytosis]], regulating neural communication in the brain. <cite>Paper1</cite> | ||
=== | === cannabimimetic hypercomputation === | ||
* | * cannabimimetic hypercomputation: a dopamine-CB1 cross-talk? | ||
** Review: [https://www.ncbi.nlm.nih.gov/pubmed/20632964 GPCR receptor heteromerization] | ** Review: [https://www.ncbi.nlm.nih.gov/pubmed/20632964 GPCR receptor heteromerization] | ||
* Heteromeric transactivation of dopamine-CB1 receptors: | * Heteromeric transactivation of dopamine-CB1 receptors: | ||
** Dopamine-CB1 heteromeric transactivation may potentiate synaptic hypercomputation in the gamma band. <cite>Paper2</cite> | ** Dopamine-CB1 heteromeric transactivation may potentiate synaptic hypercomputation in the gamma band. <cite>Paper2</cite> | ||
=== Neuronal phase coherence and synchronicity === | === Neuronal phase coherence and synchronicity === | ||
Revision as of 02:58, 1 April 2017
Hypercomputation
Synaptic hypercomputation
The Synaptic Hypercomputation (SH) hypothesis states that the phase coherence of neural communication (synaptic plasticity) may emerges via long-range synchrony in the gamma range. This non-classical neurocomputational model is controlled by synaptic exocytosis, regulating neural communication in the brain. [1]
cannabimimetic hypercomputation
- cannabimimetic hypercomputation: a dopamine-CB1 cross-talk?
- Review: GPCR receptor heteromerization
- Heteromeric transactivation of dopamine-CB1 receptors:
- Dopamine-CB1 heteromeric transactivation may potentiate synaptic hypercomputation in the gamma band. [2]
Neuronal phase coherence and synchronicity
Neuronal phase coherence is "quantum-like" entanglement because long-range synchronicity is critical for optimal communication in the gamma band. [3]
Discussion
- Is neuronal hypercomputation a form of synaptic quantum tunnelling?
- What is biological hypercomputation?
- Is self-organized criticality (SOC) an evidence of biological hypercomputation?
- What is synaptic hypercomputation?
- Is synaptic hypercomputation a function of exocytosis?
- What is biological phase coherence?
- Neuronal phase coherence and synchronicity
References
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Rhythms for Cognition: Communication through Coherence
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Concurrent Stimulation of Cannabinoid CB1 and Dopamine D2 Receptors Enhances Heterodimer Formation: A Mechanism for Receptor Cross-Talk?
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Phase-Coherence Transitions and Communication in the Gamma Range between Delay-Coupled Neuronal Populations
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Plausibility of quantum coherent states in biological systems
