User:Hua Jin: Difference between revisions

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Li, J. & Jin, H. (2007). "Regulation of brassinosteroid signaling." Trends Plant Sci 12(1): 37-41.
Li, J. & Jin, H. (2007). "Regulation of brassinosteroid signaling." Trends Plant Sci 12(1): 37-41.
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Revision as of 14:16, 4 August 2009

I am a new member of OpenWetWare!

Contact Info

Hua Jin (an artistic interpretation)
  • Hua Jin
  • Stanford University
  • 279 Campus Dr.
  • Beckman Center B-109
  • Stanford, CA 94305
  • Email me through OpenWetWare
  • I am working in Max Nachury lab at Stanford University

Education

  • 2007, PhD, University of Michigan-Ann Arbor
  • 2001, BS, Peking University, Beijing, China

Research Interest

I am interested in understand why ciliary dysfunction results in obesity in Bardet-Bidle Syndrome (BBS) patients.

Publications

Jin H, Hong Z, Su W, Li J. (2009). “A plant-specific calreticulin is a key retention factor for a defective brassinosteroid receptor in the endoplasmic reticulum. “ Proc Natl Acad Sci U S A. Jul 13. [Epub ahead of print]

Jin, H., Nachury MV. (2009). “The BBSome.” Curr Biol 19(12): R472-473

Hong, Z.*, Jin, H.*, Tzfira, T., & Li, J. (2008). "Multiple mechanism-mediated retention of a defective brassinosteroid receptor in the endoplasmic reticulum of Arabidopsis ." The Plant Cell 20: 3418-3429. (*, equal contribution)

Jin, H., Yan, Z., Nam, K. H., & Li, J. (2007). "Allele-specific suppression of a defective brassinosteroid receptor reveals a physiological role of UGGT in ER quality control." Mol Cell 26(6): 821-830.

Li, J. & Jin, H. (2007). "Regulation of brassinosteroid signaling." Trends Plant Sci 12(1): 37-41.