User:Min-Ho Kim

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*Kent State University
*Kent State University
*Kent, OH 44242, USA
*Kent, OH 44242, USA
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*[[Special:Emailuser/Min-Ho Kim|Email me through OpenWetWare]]
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*mkim15@kent.edu
==Education==
==Education==
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<!-- Feel free to add brief descriptions to your research interests as well -->
<!-- Feel free to add brief descriptions to your research interests as well -->
The major research interest in our laboratory focuses on the development of novel therapeutics to promote the resolution of non-healing chronic wounds. The research goal of our laboratory is to (1) understand the molecular and cellular mechanisms by which local inflammatory environmental cues alter phenotypic switch of tissue infiltrating innate immune cells and these cells functionally interact with endogenous or exogenously implanted mesenchymal stem cells (MSCs), and (2) develop selective strategy to engineer microenvironmental cues towards tissue regeneration by utilizing micro/nano-engineered biomaterials that enable spatio-temporal control inflammatory response. To achieve this goal, we combine interdisciplinary approaches of immuno-biology, stem cell biology, cellular and tissue engineering, and nano-bioengineering.
The major research interest in our laboratory focuses on the development of novel therapeutics to promote the resolution of non-healing chronic wounds. The research goal of our laboratory is to (1) understand the molecular and cellular mechanisms by which local inflammatory environmental cues alter phenotypic switch of tissue infiltrating innate immune cells and these cells functionally interact with endogenous or exogenously implanted mesenchymal stem cells (MSCs), and (2) develop selective strategy to engineer microenvironmental cues towards tissue regeneration by utilizing micro/nano-engineered biomaterials that enable spatio-temporal control inflammatory response. To achieve this goal, we combine interdisciplinary approaches of immuno-biology, stem cell biology, cellular and tissue engineering, and nano-bioengineering.
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==Publications==
==Publications==
<!-- Replace the PubMed ID's ("pmid=#######") below with the PubMed ID's for your publications.  You can add or remove lines as needed -->
<!-- Replace the PubMed ID's ("pmid=#######") below with the PubMed ID's for your publications.  You can add or remove lines as needed -->
<biblio>
<biblio>
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#Paper1 pmid=6947258
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#Paper1 Kim MH, Yamayoshi I, Mathew S, Lin H, Nayfach J, and Simon SI, Magnetic nanoparticle targeted hyperthermia of cutaneous S. aureus infection. Annals of Biomedical Engineering, 41:598-609, 2013
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#Paper2 pmid=13718526
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#Paper2 Dixit N, Kim MH, Leukocyte function antigen-1, kindlin-3, and calcium flux orchestrate neutrophil recruitment during Inflammation. J Immunology, 189:5954-5964, 2012
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// leave a comment about a paper here
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#Paper3 Kim MH, Granick J, Kwok C, Walker N, Borjesson DL, Curry F-E, Miller LS, and Simon SI.  Neutrophil survival and c-kit+ progenitor proliferation in Stappylococcus aureus infected skin wounds promote resolution.  Blood, 117(12):3343-3352, 2011
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#Book1 isbn=0879697164
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#Paper4 Simon SI and Kim MH, A day (or 5) in a neutrophil’s life. Blood, 116:511-512, 2010
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#Paper5 Kim MH, Curry F-E, and Simon SI. Dynamics of neutrophil extravasation and vascular permeability are uncoupled during aseptic cutaneous wounding. American Journal of Physiology-Cell Physiology, 296: C848-C856, 2009
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#Paper6 Kim MH, Liu W, Borjesson DL, Curry F-E, Miller LS, Cheung AL, Liu F-T, Isseroff RR, and Simon SI. Dynamics of neutrophil infiltration during cutaneous wound healing and infection using fluorescence imaging. Journal of Investigative Dermatology. 128: 1812-1820, 2008
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#Paper7 Kim MH, Granger DN, and Harris NR. Mediators of CD18/P-selectin-dependent constriction of venule-paired arterioles in hypercholesterolemia. Microvascular Research, 73, pp. 150-155, 2007
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#Paper8 Kim MH, Carter P, and Harris NR. P-selectin-mediated adhesion impairs endothelium-dependent arteriolar dilation in hypercholesterolemic mice. American Journal of Physiology -Heart and Circ Physiology. 292: H632-H638, 2007 
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#Paper9 Kim MH and Harris NR. Leukocyte adherence inhibits adenosine-dependent venular control of arteriolar diameter and nitric oxide. American Journal of Physiology (Heart and Circ Physiology). 291: H724-H731, 2006 
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#Paper10 Kim MH, Harris NR and Tarbell JM. Regulation of hydraulic conductivity in response to sustained changes in pressure. American Journal of Physiology (Heart and Circ Physiology). 289: H2551-H2558, 2005
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#Paper11 Kim MH, Harris NR and Tarbell JM. Regulation of capillary hydraulic conductivity in response to acute change in shear.  American Journal of Physiology (Heart and Circ Physiology), 289: H2126-H2135, 2005
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#Paper12 Kim MH, Harris NR and Tarbell JM. Control of the arteriolar myogenic response by transvascular fluid filtration. Microvascular Research, 68, pp. 30-37, 2004 
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#Paper13 Cho M, Kim MH & et all. A Study on the Room-Temperature Curvature Shapes of Unsymmetric Laminates Including Slippage Effects. Journal of Composite Materials, Vol.32,  No. 5, pp. 460 - 482, 1998
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#Paper14 Cho M, Kim K and Kim MH. Efficient Higher-order Shell Theory for Laminated Composites. Composite Structures, 34, pp. 197 - 212, 1996
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#Paper15 Cho M and Kim MH. A Postprocess Method Using a Displacement Field of Higher Shell Theory. Composite Structures, 34, pp. 185 - 196, 1996
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</biblio>
</biblio>

Current revision

I am a new member of OpenWetWare!

Contents

Contact Info

Min-Ho Kim (an artistic interpretation)
Min-Ho Kim (an artistic interpretation)
  • Min-Ho Kim, Ph.D
  • Assistant Professor of Biological Sciences
  • Graduate Faculty, School of Biomedical Sciences
  • College of Arts and Sciences
  • Kent State University
  • Kent, OH 44242, USA
  • mkim15@kent.edu

Education

  • 2004, PhD, Bioengineering, Pennsylvania State University
  • 1996, ME, Inha University, South Korea
  • 1994, BE, Inha University, South Korea

Research interests

The major research interest in our laboratory focuses on the development of novel therapeutics to promote the resolution of non-healing chronic wounds. The research goal of our laboratory is to (1) understand the molecular and cellular mechanisms by which local inflammatory environmental cues alter phenotypic switch of tissue infiltrating innate immune cells and these cells functionally interact with endogenous or exogenously implanted mesenchymal stem cells (MSCs), and (2) develop selective strategy to engineer microenvironmental cues towards tissue regeneration by utilizing micro/nano-engineered biomaterials that enable spatio-temporal control inflammatory response. To achieve this goal, we combine interdisciplinary approaches of immuno-biology, stem cell biology, cellular and tissue engineering, and nano-bioengineering.

Publications

  1. Kim MH, Yamayoshi I, Mathew S, Lin H, Nayfach J, and Simon SI, Magnetic nanoparticle targeted hyperthermia of cutaneous S. aureus infection. Annals of Biomedical Engineering, 41:598-609, 2013 [Paper1]
  2. Dixit N, Kim MH, Leukocyte function antigen-1, kindlin-3, and calcium flux orchestrate neutrophil recruitment during Inflammation. J Immunology, 189:5954-5964, 2012 [Paper2]
  3. Kim MH, Granick J, Kwok C, Walker N, Borjesson DL, Curry F-E, Miller LS, and Simon SI. Neutrophil survival and c-kit+ progenitor proliferation in Stappylococcus aureus infected skin wounds promote resolution. Blood, 117(12):3343-3352, 2011 [Paper3]
  4. Simon SI and Kim MH, A day (or 5) in a neutrophil’s life. Blood, 116:511-512, 2010 [Paper4]
  5. Kim MH, Curry F-E, and Simon SI. Dynamics of neutrophil extravasation and vascular permeability are uncoupled during aseptic cutaneous wounding. American Journal of Physiology-Cell Physiology, 296: C848-C856, 2009 [Paper5]
  6. Kim MH, Liu W, Borjesson DL, Curry F-E, Miller LS, Cheung AL, Liu F-T, Isseroff RR, and Simon SI. Dynamics of neutrophil infiltration during cutaneous wound healing and infection using fluorescence imaging. Journal of Investigative Dermatology. 128: 1812-1820, 2008 [Paper6]
  7. Kim MH, Granger DN, and Harris NR. Mediators of CD18/P-selectin-dependent constriction of venule-paired arterioles in hypercholesterolemia. Microvascular Research, 73, pp. 150-155, 2007 [Paper7]
  8. Kim MH, Carter P, and Harris NR. P-selectin-mediated adhesion impairs endothelium-dependent arteriolar dilation in hypercholesterolemic mice. American Journal of Physiology -Heart and Circ Physiology. 292: H632-H638, 2007 [Paper8]
  9. Kim MH and Harris NR. Leukocyte adherence inhibits adenosine-dependent venular control of arteriolar diameter and nitric oxide. American Journal of Physiology (Heart and Circ Physiology). 291: H724-H731, 2006 [Paper9]
  10. Kim MH, Harris NR and Tarbell JM. Regulation of hydraulic conductivity in response to sustained changes in pressure. American Journal of Physiology (Heart and Circ Physiology). 289: H2551-H2558, 2005 [Paper10]
  11. Kim MH, Harris NR and Tarbell JM. Regulation of capillary hydraulic conductivity in response to acute change in shear. American Journal of Physiology (Heart and Circ Physiology), 289: H2126-H2135, 2005 [Paper11]
  12. Kim MH, Harris NR and Tarbell JM. Control of the arteriolar myogenic response by transvascular fluid filtration. Microvascular Research, 68, pp. 30-37, 2004 [Paper12]
  13. Cho M, Kim MH & et all. A Study on the Room-Temperature Curvature Shapes of Unsymmetric Laminates Including Slippage Effects. Journal of Composite Materials, Vol.32, No. 5, pp. 460 - 482, 1998 [Paper13]
  14. Cho M, Kim K and Kim MH. Efficient Higher-order Shell Theory for Laminated Composites. Composite Structures, 34, pp. 197 - 212, 1996 [Paper14]
  15. Cho M and Kim MH. A Postprocess Method Using a Displacement Field of Higher Shell Theory. Composite Structures, 34, pp. 185 - 196, 1996 [Paper15]

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