User:Nizar Batada: Difference between revisions

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<tr><td>2004--2006</td><td>Postdoctoral fellow,<br>
<tr><td>2004--2006</td><td>Postdoctoral fellow,<br>
Department of Medical Genetics, University of Toronto([http://www.mshri.on.ca/tyers/ Mike Tyers], advisor) and<br> Department of Biology, University of Bath ([http://www.bath.ac.uk/bio-sci/research/profiles/hurst-l.html Laurence Hurst], advisor)</td></tr>
Department of Medical Genetics, University of Toronto ([http://www.mshri.on.ca/tyers/ Mike Tyers], advisor) and<br> Department of Biology, University of Bath ([http://www.bath.ac.uk/bio-sci/research/profiles/hurst-l.html Laurence Hurst], advisor)</td></tr>
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<td>2004</td><td>PhD in Biophysics,<br>
<td>2004</td><td>PhD in Biophysics,<br>
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# Computational analysis of epigenetics modification patterns
# Computational analysis of epigenetics modification patterns
# Genome organization and evolution
# Genome organization and evolution
[http://openwetware.org/index.php?title=Batada_Lab Lab Homepage]

Revision as of 20:55, 29 August 2008

Contact Info

Nizar Batada

Nizar Batada
Ontario Institute of Cancer Research
101 College St, Suite 800,
MaRS Center, South Tower,
Toronto, Ontario M5G 3A0

tel: 416-673-8594
fax: 416-977-1118
http://www.oicr.on.ca/research/batada.htm

Administrative Assistant:
Stacey Quinn
tel: 416-673-8519
stacey.quinn "at" oicr.on.ca

Biography

2008-- Principal Investigator,
Cancer Genomics, Ontario Institute for Cancer Research
2007--2008Postdoctoral fellow,
Department of Genetics, Harvard Medical School (George Church, advisor)
2004--2006Postdoctoral fellow,
Department of Medical Genetics, University of Toronto (Mike Tyers, advisor) and
Department of Biology, University of Bath (Laurence Hurst, advisor)
2004PhD in Biophysics,
Stanford University (Michael Levitt, advisor)

Research interests

  1. Understand how chromatin states relate to gene expression
  2. How epigenomes of various stem cells change during differentiation; and how the characteristic tissue-specific epigenomes change during reprogramming
  3. How chromatin modification (DNA methylation and histone modification) are established and maintained; in particular identification of genomic features that allow prediction of epigenetically labile genes
  4. Contribution of chromatin state to variability or "noise" in transcription
  5. Computational analysis of epigenetics modification patterns
  6. Genome organization and evolution