User:Orsolya Kiraly

From OpenWetWare

(Difference between revisions)
Jump to: navigation, search
Line 1: Line 1:
-
<!-- Delete this entire line as part of your first edit of your user page --> {{New user}}
 
==Contact Info==
==Contact Info==
Line 5: Line 4:
*Orsolya Kiraly
*Orsolya Kiraly
-
*MIT
+
*Postdoctoral Associate
-
*Address 1
+
*Engelward laboratory
-
*Address 2
+
*Department of Biological Engineering
-
*City, State, Country etc.
+
*Massachusetts Institute of Technology
 +
*77 Massachusetts Ave
 +
*Lab: 617-750-7335 (Room 16-760)
 +
*Cambridge, MA
*[[Special:Emailuser/Orsolya Kiraly|Email me through OpenWetWare]]
*[[Special:Emailuser/Orsolya Kiraly|Email me through OpenWetWare]]
-
I work in the [[Your Lab]] at XYZ UniversityI learned about [[OpenWetWare]] from 20.109 instructors, and I've joined because Teaching 20.109.
+
I received my PhD for work on how rare mutations in pancreatic trypsin inhibitor contribute to chronic pancreatic inflammation.  In the [http://web.mit.edu/engelward-lab/], my current work is aimed at homologous recombination in the pancreas in vivo.
 +
 
 +
While mitotic homologous recombination is an important DNA repair/tolerance mechanism, it can result in sequence rearrangements that can contribute to cancer. I am investigating the effects of DNA damaging chemicals, radiation, cell proliferation and DNA repair on homologous recombination in the pancreas.  
 +
 
 +
The next question in my project is whether homologous recombination is induced by inflammation, which is a major risk factor for cancer.
 +
 
==Education==
==Education==

Revision as of 10:01, 1 September 2010

Contents

Contact Info

Orsolya Kiraly (an artistic interpretation)
Orsolya Kiraly (an artistic interpretation)
  • Orsolya Kiraly
  • Postdoctoral Associate
  • Engelward laboratory
  • Department of Biological Engineering
  • Massachusetts Institute of Technology
  • 77 Massachusetts Ave
  • Lab: 617-750-7335 (Room 16-760)
  • Cambridge, MA
  • Email me through OpenWetWare

I received my PhD for work on how rare mutations in pancreatic trypsin inhibitor contribute to chronic pancreatic inflammation. In the [1], my current work is aimed at homologous recombination in the pancreas in vivo.

While mitotic homologous recombination is an important DNA repair/tolerance mechanism, it can result in sequence rearrangements that can contribute to cancer. I am investigating the effects of DNA damaging chemicals, radiation, cell proliferation and DNA repair on homologous recombination in the pancreas.

The next question in my project is whether homologous recombination is induced by inflammation, which is a major risk factor for cancer.


Education

  • Year, PhD, Institute
  • Year, MS, Institute
  • Year, BS, Institute

Research interests

  1. Interest 1
  2. Interest 2
  3. Interest 3

Publications

  1. Goldbeter A and Koshland DE Jr. . pmid:6947258. PubMed HubMed [Paper1]
  2. JACOB F and MONOD J. . pmid:13718526. PubMed HubMed [Paper2]
    leave a comment about a paper here

  3. Mark Ptashne. A genetic switch. Cold Spring Harbor, N.Y.: Cold Spring Harbor Laboratory Press, 2004. isbn:0879697164. [Book1]
All Medline abstracts: PubMed HubMed

Useful links

Personal tools