User:Shannon K. Alford

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==My Teaching Goals==
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Hands-on experience at the bench is useful for all engineers; experimentalists and theoreticians alike. In biological engineering, understanding how to experimentally perturb cell-based systems in a systematic and controlled manner is paramount to applying an engineering perspective across disciplines. My aim as an instructor of 20.109 is to provide practical, interesting, and motivating experiences that promote rewarding Aha! moments and drive novel insight and innovation. I believe that mentoring at the personal level, as well as the instructional level, is key to preparing the next generation of bioengineers for their careers tackling global technical health challenges. As such, I strive to provide opportunities for all students to improve and succeed in performing and communicating their science.
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==My Research interests==
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==My Research Interests==
Aberrant cell migration is a hallmark of several invasive diseases, such as metastatic cancer and systemic autoimmune disorders. I am particularly interested in the regulation of cell motility and the underlying intracellular signaling processes as modulated by interaction with the cytoskeleton. My current research combines biochemical, cell biological and systems biological engineering approaches to elucidate the intracellular signaling mechanism underlying increased breast cancer metastasis due to expression of an invasion-specific protein, Mena<sup>INV</sup>.
Aberrant cell migration is a hallmark of several invasive diseases, such as metastatic cancer and systemic autoimmune disorders. I am particularly interested in the regulation of cell motility and the underlying intracellular signaling processes as modulated by interaction with the cytoskeleton. My current research combines biochemical, cell biological and systems biological engineering approaches to elucidate the intracellular signaling mechanism underlying increased breast cancer metastasis due to expression of an invasion-specific protein, Mena<sup>INV</sup>.
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==Publications==
 
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<!-- Replace the PubMed ID's ("pmid=#######") below with the PubMed ID's for your publications.  You can add or remove lines as needed -->
 
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<biblio>
 
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#Paper1 pmid=6947258
 
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#Paper2 pmid=13718526
 
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// leave a comment about a paper here
 
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#Book1 isbn=0879697164
 
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</biblio>
 
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==Useful links==
 
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*[[OpenWetWare:Welcome|Introductory tutorial]]
 
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*[[Help|OpenWetWare help pages]]
 

Revision as of 11:31, 31 August 2012

I am a new member of OpenWetWare!

Contact Info

Shannon Hughes-Alford
Shannon Hughes-Alford

Biological Engineering 20.109 Instructor & Research Scientist

  • Office: Bldg 56-389
  • Lab: Blgd 56-378
  • Office Phone: 617-258-9488
  • Email: skalford AT mit DOT edu
Butterstick, the office motivator
Butterstick, the office motivator


My Teaching Goals

Hands-on experience at the bench is useful for all engineers; experimentalists and theoreticians alike. In biological engineering, understanding how to experimentally perturb cell-based systems in a systematic and controlled manner is paramount to applying an engineering perspective across disciplines. My aim as an instructor of 20.109 is to provide practical, interesting, and motivating experiences that promote rewarding Aha! moments and drive novel insight and innovation. I believe that mentoring at the personal level, as well as the instructional level, is key to preparing the next generation of bioengineers for their careers tackling global technical health challenges. As such, I strive to provide opportunities for all students to improve and succeed in performing and communicating their science.


My Research Interests

Aberrant cell migration is a hallmark of several invasive diseases, such as metastatic cancer and systemic autoimmune disorders. I am particularly interested in the regulation of cell motility and the underlying intracellular signaling processes as modulated by interaction with the cytoskeleton. My current research combines biochemical, cell biological and systems biological engineering approaches to elucidate the intracellular signaling mechanism underlying increased breast cancer metastasis due to expression of an invasion-specific protein, MenaINV.

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