User:Tkadm30/Notebook/Hypercomputation
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Hypercomputation
Synaptic hypercomputation
The Synaptic Hypercomputation (SH) hypothesis states that the phase coherence of neural communication (synaptic plasticity) may emerges via long-range synchrony in the gamma range. This non-classical neurocomputational model is controlled by synaptic exocytosis, regulating neural communication in the brain. [1]
Pharmacological hypercomputation
- Is pharmacological hypercomputation (PH) a dopamine-mediated synaptic function? (exocytosis)
- THC may rewire the brain connectivity by enhancing functional NMDA expression. (Reference needed)
Neuronal phase coherence and synaptic transitions
Heteromeric transactivation of dopamine-CB1 receptors:
- How dopamine-CB1 heteromeric transactivation potentiate synaptic hypercomputation in the gamma band? [2]
Discussion
- Is neuronal hypercomputation a form of synaptic quantum tunnelling?
- What is biological hypercomputation?
- Is self-organized criticality (SOC) an evidence of biological hypercomputation?
- What is synaptic hypercomputation?
- What is biological phase coherence?
References
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Rhythms for Cognition: Communication through Coherence
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Concurrent Stimulation of Cannabinoid CB1 and Dopamine D2 Receptors Enhances Heterodimer Formation: A Mechanism for Receptor Cross-Talk?
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Important contribution of alpha-neurexins to Ca2+-triggered exocytosis of secretory granules.