User:Tkadm30/Notebook/Hypercomputation: Difference between revisions

From OpenWetWare
Jump to navigationJump to search
No edit summary
Line 11: Line 11:
* Is pharmacological hypercomputation (PH) a dopamine-mediated synaptic function? (exocytosis)  
* Is pharmacological hypercomputation (PH) a dopamine-mediated synaptic function? (exocytosis)  
* Cannabinoids may rewire the brain connectivity by enhancing functional NMDA expression and [http://www.thenakedscientists.com/forum/index.php?topic=68779 cognitive intelligence].
* Cannabinoids may rewire the brain connectivity by enhancing functional NMDA expression and [http://www.thenakedscientists.com/forum/index.php?topic=68779 cognitive intelligence].
== Neuronal phase coherence and transitions ==


=== Heteromeric transactivation of adenosine-CB1 receptors ===
=== Heteromeric transactivation of adenosine-CB1 receptors ===

Revision as of 21:28, 13 January 2017

Hypercomputation

Synaptic hypercomputation

The Synaptic Hypercomputation (SH) hypothesis states that the phase coherence of neural communication may emerges via synaptic connectivity. This non-classical neurocomputational model is controlled by synaptic exocytosis, regulating neural synchronicity in the CNS.

Pharmacological hypercomputation

  • Is pharmacological hypercomputation (PH) a dopamine-mediated synaptic function? (exocytosis)
  • Cannabinoids may rewire the brain connectivity by enhancing functional NMDA expression and cognitive intelligence.

Neuronal phase coherence and transitions

Heteromeric transactivation of adenosine-CB1 receptors

How adenosine-CB1 heteromeric transactivation potentiate synaptic hypercomputation?

Neurexin-mediated trans-synaptic exocytosis

Could retrograde signaling promote synaptic proteins synthesis ? https://www.ncbi.nlm.nih.gov/pubmed/17035546

Discussion

See also