User:Tkadm30/Notebook/Hypercomputation: Difference between revisions
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#Paper2 http://molpharm.aspetjournals.org/content/67/5/1697.short | #Paper2 http://molpharm.aspetjournals.org/content/67/5/1697.short | ||
//Concurrent Stimulation of Cannabinoid CB1 and Dopamine D2 Receptors Enhances Heterodimer Formation: A Mechanism for Receptor Cross-Talk? | //Concurrent Stimulation of Cannabinoid CB1 and Dopamine D2 Receptors Enhances Heterodimer Formation: A Mechanism for Receptor Cross-Talk? | ||
</biblio> | </biblio> | ||
== See also == | == See also == | ||
* [[User:Etienne_Robillard/Notebook/Endocannabinoids|Endocannabinoids Notebook]] | * [[User:Etienne_Robillard/Notebook/Endocannabinoids|Endocannabinoids Notebook]] |
Revision as of 03:52, 25 January 2017
Hypercomputation
Synaptic hypercomputation
The Synaptic Hypercomputation (SH) hypothesis states that the phase coherence of neural communication (synaptic plasticity) may emerges via long-range synchrony in the gamma range. This non-classical neurocomputational model is controlled by synaptic exocytosis, regulating neural communication in the brain. [1]
Pharmacological hypercomputation
- Is pharmacological hypercomputation (PH) a dopamine-mediated synaptic function? (exocytosis)
- THC may rewire the brain connectivity by enhancing functional NMDA expression. (Reference needed)
Neuronal phase coherence and synaptic transitions
Heteromeric transactivation of dopamine-CB1 receptors:
- How dopamine-CB1 heteromeric transactivation potentiate synaptic hypercomputation in the gamma band? [2]
Discussion
- Is neuronal hypercomputation a form of synaptic quantum tunnelling?
- What is biological hypercomputation?
- Is self-organized criticality (SOC) an evidence of biological hypercomputation?
- What is synaptic hypercomputation?
- Is synaptic hypercomputation a function of exocytosis?
- What is biological phase coherence?
References
-
Rhythms for Cognition: Communication through Coherence
-
Concurrent Stimulation of Cannabinoid CB1 and Dopamine D2 Receptors Enhances Heterodimer Formation: A Mechanism for Receptor Cross-Talk?